Abscisic acid
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| Category | Others |
| Catalog number | BBF-03830 |
| CAS | 14375-45-2 |
| Molecular Weight | 264.32 |
| Molecular Formula | C15H20O4 |
| Purity | ≥ 98 % |
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Description
A plant hormone involved in many cellular processes such as stomatal closure, water and ion uptake control, leaf abscission and senescence.
Specification
| Synonyms | Dormin |
| Storage | Store at -20 ℃ |
| IUPAC Name | (2Z,4E)-5-(1-hydroxy-2,6,6-trimethyl-4-oxocyclohex-2-en-1-yl)-3-methylpenta-2,4-dienoic acid |
| Canonical SMILES | CC1=CC(=O)CC(C1(C=CC(=CC(=O)O)C)O)(C)C |
| InChI | InChI=1S/C15H20O4/c1-10(7-13(17)18)5-6-15(19)11(2)8-12(16)9-14(15,3)4/h5-8,19H,9H2,1-4H3,(H,17,18)/b6-5+,10-7- |
| InChI Key | JLIDBLDQVAYHNE-LXGGSRJLSA-N |
Properties
| Appearance | White powder |
| Boiling Point | 458.7 ℃ at 760 mmHg |
| Melting Point | 186-188 ℃(lit.) |
| Density | 1.193 g/cm3 |
| LogP | 2.24990 |
Reference Reading
1.Regulatory Networks in Pollen Development under Cold Stress.
Sharma KD1, Nayyar H2. Front Plant Sci. 2016 Mar 31;7:402. doi: 10.3389/fpls.2016.00402. eCollection 2016.
Cold stress modifies anthers' metabolic pathways to induce pollen sterility. Cold-tolerant plants, unlike the susceptible ones, produce high proportion of viable pollen. Anthers in susceptible plants, when exposed to cold stress, increase abscisic acid (ABA) metabolism and reduce ABA catabolism. Increased ABA negatively regulates expression of tapetum cell wall bound invertase and monosaccharide transport genes resulting in distorted carbohydrate pool in anther. Cold-stress also reduces endogenous levels of the bioactive gibberellins (GAs), GA4 and GA7, in susceptible anthers by repression of the GA biosynthesis genes. Here, we discuss recent findings on mechanisms of cold susceptibility in anthers which determine pollen sterility. We also discuss differences in regulatory pathways between cold-stressed anthers of susceptible and tolerant plants that decide pollen sterility or viability.
2.Differential Activation of the Wheat SnRK2 Family by Abiotic Stresses.
Zhang H1, Li W2, Mao X3, Jing R3, Jia H1. Front Plant Sci. 2016 Mar 31;7:420. doi: 10.3389/fpls.2016.00420. eCollection 2016.
Plant responses to stress occur via abscisic acid (ABA) dependent or independent pathways. Sucrose non-fermenting1-related protein kinase 2 (SnRK2) play a key role in plant stress signal transduction pathways. It is known that some SnRK2 members are positive regulators of ABA signal transduction through interaction with group A type 2C protein phosphatases (PP2Cs). Here, 10 SnRK2s were isolated from wheat. Based on phylogenetic analysis using kinase domains or the C-terminus, the 10 SnRK2s were divided into three subclasses. Expression pattern analysis revealed that all TaSnRK2s were involved in the responses to PEG, NaCl, and cold stress. TaSnRK2s in subclass III were strongly induced by ABA. Subclass II TaSnRK2s responded weakly to ABA, whereas TaSnRK2s in subclass I were not activated by ABA treatment. Motif scanning in the C-terminus indicated that motifs 4 and 5 in the C-terminus were unique to subclass III. We further demonstrate the physical and functional interaction between TaSnRK2s and a typical group A PP2C (TaABI1) using Y2H and BiFC assays.
3.Dissecting abscisic acid signaling pathways involved in cuticle formation.
Cui F1, Brosché M2, Lehtonen MT3, Amiryousefi A4, Xu E4, Punkkinen M5, Valkonen JP3, Fujii H5, Overmyer K4. Mol Plant. 2016 Apr 6. pii: S1674-2052(16)30022-3. doi: 10.1016/j.molp.2016.04.001. [Epub ahead of print]
The cuticle is the outer physical barrier of aerial plant surfaces and an important interaction point between plants and the environment. Many environmental stresses affect cuticle formation, yet the regulatory pathways involved remain undefined. We used a genetics and gene expression approach in Arabidopsis thaliana to define an abscisic acid (ABA) signaling loop that positively regulated cuticle formation via the core ABA signaling pathway, PYR/PYL receptors, PP2C phosphatase and SNF1-Related Protein Kinase (SnRK) 2.2/ SnRK2.3/ SnRK2.6. Downstream from the SnRK2 kinases, cuticle formation was not regulated by the ABA Responsive Element-Binding transcription factors (TFs), rather by DEWAX, MYB16, MYB94 and MYB96. Additionally, low air humidity increased cuticle formation independent of the core ABA pathway and cell death/ROS signaling attenuated expression of cuticle-biosynthesis genes. In Physcomitrella patens, exogenous ABA suppressed expression of cuticle-related genes, whose Arabidopsis orthologues were ABA-induced.
4.Isolation and functional characterization of a cold responsive phosphatidylinositol transfer-associated protein, ZmSEC14p, from maize (Zea may L.).
Wang X1, Shan X1, Xue C1, Wu Y1, Su S1, Li S1, Liu H1, Jiang Y1, Zhang Y1, Yuan Y2. Plant Cell Rep. 2016 Apr 9. [Epub ahead of print]
KEY MESSAGE: A Sec14-like protein, ZmSEC14p , from maize was structurally analyzed and functionally tested. Overexpression of ZmSEC14p in transgenic Arabidopsis conferred tolerance to cold stress. Sec14-like proteins are involved in essential biological processes, such as phospholipid metabolism, signal transduction, membrane trafficking, and stress response. Here, we reported a phosphatidylinositol transfer-associated protein, ZmSEC14p (accession no. KT932998), isolated from a cold-tolerant maize inbred line using the cDNA-AFLP approach and RACE-PCR method. Full-length cDNA that consisted of a single open reading frame (ORF) encoded a putative polypeptide of 295 amino acids. The ZmSEC14p protein was mainly localized in the nucleus, and its transcript was induced by cold, salt stresses, and abscisic acid (ABA) treatment in maize leaves and roots. Overexpression of ZmSEC14p in transgenic Arabidopsis conferred tolerance to cold stress. This tolerance was primarily displayed by the increased germination rate, root length, plant survival rate, accumulation of proline, activities of antioxidant enzymes, and the reduction of oxidative damage by reactive oxygen species (ROS).
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Bio Calculators
* Our calculator is based on the following equation:
Concentration (start) x Volume (start) = Concentration (final) x Volume (final)
It is commonly abbreviated as: C1V1 = C2V2
* Total Molecular Weight:
g/mol
Tip: Chemical formula is case sensitive. C22H30N4O √ c22h30n40 ╳
